by Sherree Gray, Martin
Christensen, Judy Craft
Intensive and Critical Care Nursing
Intra-abdominal
hypertension is classified as either primary or secondary – primary occurs due
to intra-abdominal or retro-peritoneal pathophysiology, whereas secondary
results in alterations in capillary fluid dynamics due to factors, such as
massive fluid resuscitation and generalised inflammation. The renal and
gastro-intestinal effects occur early in the progression of intra-abdominal
hypertension, and may lead to poor patient outcomes if not identified. As a
direct response to intra-abdominal hypertension, renal function is reduced with
remarkable impairment from pressures of around 10 mmHg, oliguria developing at
15 mmHg and anuria developing at 30 mmHg.
